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lignoceric acid  (MedChemExpress)


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    MedChemExpress lignoceric acid
    Lignoceric Acid, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 93/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/lignoceric+acid/Lignoceric+acid/pmc12175649-117-8-32
    Average 93 stars, based on 2 article reviews
    lignoceric acid - by Bioz Stars, 2026-09
    93/100 stars

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    other:

    Article Title: Targeting ABCD1-ACOX1-MET/IGF1R axis suppresses multiple myeloma
    Article Snippet: Carfilzomib (HY-10455), lenalidomide (HY-A0003), 10,12-tricosadiynoic acid (TDYA (HY-135425)), lignoceric acid (C24:0; HY-121883), nervonic acid (C24:1; HY-N2526), hexacosanoic acid (C26:0; HY-113301), arachidic acid (C20:0; HY-W004260), and eicosenoic acid (C20:1; HY-109589A) were purchased from MedChemExpress LLC (Monmouth Junction, NJ, USA).

    Article Title: Exploring the mechanism of Paotianxiong polysaccharide in the treatment of chronic kidney disease combining metabolomics and microbiomics technologies.
    Article Snippet: A close relationship between the pathogenesis of chronic kidney disease (CKD) and abnormalities in the gutkidney axis.. Paotianxiong polysaccharides (PTXP) that have demonstrated therapeutic effects on CKD.. However, the specific mechanism by which PTXP ameliorates CKD through the gut-kidney axis remains to be explored.



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    A Schematic created using BioRender illustrating PISD enzyme reaction with phosphatidylserine (PS) to form CO 2 and phosphatidylethanolamine (PE). B Schematic created using BioRender showing generation of shScrambled (shSCR) and shPISD lentivirus in 293T cells and subsequent infection and gene silencing in HEPA1-6 cells. C The shPISD lentivirus reduced PISD mRNA abundance ( n = 2 per group) and altered the abundance of various genes involved in phospholipid metabolism ( n = 5 per group). D PISD activity was reduced in shPISD treated cells ( n = 3 per group). E , F Thin layer chromatography images indicating shPISD treated cells had reduced mitochondrial PE content ( E ) and densitometry quantification of thin layer chromatography images presented in E ( n = 3 per group) ( F ). G A Seahorse mitochondrial stress test reveals shPISD treated cells have reduced FCCP stimulated respiration rates ( n = 10 per group). H shPISD treated cells have elevated proton leak, calculated by subtracting oxygen consumption rates (OCR) during basal conditions from OCR during oligomycin (oligo) treated conditions ( n = 10 per group). I , J Western blot image indicating shPISD treated cells have reduced complex I and IV protein abundance ( I ) and densitometry quantification of Western blot images shown in I ( n = 6 per group) ( J ). K 1- 14 C oleate oxidation is reduced in shPISD treated cells ( n = 6 per group). L 1- 14 C <t>lignoceric</t> acid oxidation is increased in shPISD treated cells ( n = 6 per group). M PCA plot of untargeted metabolomics data illustrating that shPISD treated cells have an altered metabolome compared to shSCR treated cells ( n = 3 per group). N Volcano plot of untargeted metabolomics data illustrating differences in metabolites between shSCR and shPISD treated cells ( n = 3 per group). O Heatmap of select lipid metabolism related metabolites that were differentially expressed between shSCR and shPISD treated cells ( n = 3 per group). The data are presented as means ± S.D.
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    A Schematic created using BioRender illustrating PISD enzyme reaction with phosphatidylserine (PS) to form CO 2 and phosphatidylethanolamine (PE). B Schematic created using BioRender showing generation of shScrambled (shSCR) and shPISD lentivirus in 293T cells and subsequent infection and gene silencing in HEPA1-6 cells. C The shPISD lentivirus reduced PISD mRNA abundance ( n = 2 per group) and altered the abundance of various genes involved in phospholipid metabolism ( n = 5 per group). D PISD activity was reduced in shPISD treated cells ( n = 3 per group). E , F Thin layer chromatography images indicating shPISD treated cells had reduced mitochondrial PE content ( E ) and densitometry quantification of thin layer chromatography images presented in E ( n = 3 per group) ( F ). G A Seahorse mitochondrial stress test reveals shPISD treated cells have reduced FCCP stimulated respiration rates ( n = 10 per group). H shPISD treated cells have elevated proton leak, calculated by subtracting oxygen consumption rates (OCR) during basal conditions from OCR during oligomycin (oligo) treated conditions ( n = 10 per group). I , J Western blot image indicating shPISD treated cells have reduced complex I and IV protein abundance ( I ) and densitometry quantification of Western blot images shown in I ( n = 6 per group) ( J ). K 1- 14 C oleate oxidation is reduced in shPISD treated cells ( n = 6 per group). L 1- 14 C <t>lignoceric</t> acid oxidation is increased in shPISD treated cells ( n = 6 per group). M PCA plot of untargeted metabolomics data illustrating that shPISD treated cells have an altered metabolome compared to shSCR treated cells ( n = 3 per group). N Volcano plot of untargeted metabolomics data illustrating differences in metabolites between shSCR and shPISD treated cells ( n = 3 per group). O Heatmap of select lipid metabolism related metabolites that were differentially expressed between shSCR and shPISD treated cells ( n = 3 per group). The data are presented as means ± S.D.
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    A Schematic created using BioRender illustrating PISD enzyme reaction with phosphatidylserine (PS) to form CO 2 and phosphatidylethanolamine (PE). B Schematic created using BioRender showing generation of shScrambled (shSCR) and shPISD lentivirus in 293T cells and subsequent infection and gene silencing in HEPA1-6 cells. C The shPISD lentivirus reduced PISD mRNA abundance ( n = 2 per group) and altered the abundance of various genes involved in phospholipid metabolism ( n = 5 per group). D PISD activity was reduced in shPISD treated cells ( n = 3 per group). E , F Thin layer chromatography images indicating shPISD treated cells had reduced mitochondrial PE content ( E ) and densitometry quantification of thin layer chromatography images presented in E ( n = 3 per group) ( F ). G A Seahorse mitochondrial stress test reveals shPISD treated cells have reduced FCCP stimulated respiration rates ( n = 10 per group). H shPISD treated cells have elevated proton leak, calculated by subtracting oxygen consumption rates (OCR) during basal conditions from OCR during oligomycin (oligo) treated conditions ( n = 10 per group). I , J Western blot image indicating shPISD treated cells have reduced complex I and IV protein abundance ( I ) and densitometry quantification of Western blot images shown in I ( n = 6 per group) ( J ). K 1- 14 C oleate oxidation is reduced in shPISD treated cells ( n = 6 per group). L 1- 14 C <t>lignoceric</t> acid oxidation is increased in shPISD treated cells ( n = 6 per group). M PCA plot of untargeted metabolomics data illustrating that shPISD treated cells have an altered metabolome compared to shSCR treated cells ( n = 3 per group). N Volcano plot of untargeted metabolomics data illustrating differences in metabolites between shSCR and shPISD treated cells ( n = 3 per group). O Heatmap of select lipid metabolism related metabolites that were differentially expressed between shSCR and shPISD treated cells ( n = 3 per group). The data are presented as means ± S.D.
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    A Schematic created using BioRender illustrating PISD enzyme reaction with phosphatidylserine (PS) to form CO 2 and phosphatidylethanolamine (PE). B Schematic created using BioRender showing generation of shScrambled (shSCR) and shPISD lentivirus in 293T cells and subsequent infection and gene silencing in HEPA1-6 cells. C The shPISD lentivirus reduced PISD mRNA abundance ( n = 2 per group) and altered the abundance of various genes involved in phospholipid metabolism ( n = 5 per group). D PISD activity was reduced in shPISD treated cells ( n = 3 per group). E , F Thin layer chromatography images indicating shPISD treated cells had reduced mitochondrial PE content ( E ) and densitometry quantification of thin layer chromatography images presented in E ( n = 3 per group) ( F ). G A Seahorse mitochondrial stress test reveals shPISD treated cells have reduced FCCP stimulated respiration rates ( n = 10 per group). H shPISD treated cells have elevated proton leak, calculated by subtracting oxygen consumption rates (OCR) during basal conditions from OCR during oligomycin (oligo) treated conditions ( n = 10 per group). I , J Western blot image indicating shPISD treated cells have reduced complex I and IV protein abundance ( I ) and densitometry quantification of Western blot images shown in I ( n = 6 per group) ( J ). K 1- 14 C oleate oxidation is reduced in shPISD treated cells ( n = 6 per group). L 1- 14 C <t>lignoceric</t> acid oxidation is increased in shPISD treated cells ( n = 6 per group). M PCA plot of untargeted metabolomics data illustrating that shPISD treated cells have an altered metabolome compared to shSCR treated cells ( n = 3 per group). N Volcano plot of untargeted metabolomics data illustrating differences in metabolites between shSCR and shPISD treated cells ( n = 3 per group). O Heatmap of select lipid metabolism related metabolites that were differentially expressed between shSCR and shPISD treated cells ( n = 3 per group). The data are presented as means ± S.D.
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    Image Search Results


    A Schematic created using BioRender illustrating PISD enzyme reaction with phosphatidylserine (PS) to form CO 2 and phosphatidylethanolamine (PE). B Schematic created using BioRender showing generation of shScrambled (shSCR) and shPISD lentivirus in 293T cells and subsequent infection and gene silencing in HEPA1-6 cells. C The shPISD lentivirus reduced PISD mRNA abundance ( n = 2 per group) and altered the abundance of various genes involved in phospholipid metabolism ( n = 5 per group). D PISD activity was reduced in shPISD treated cells ( n = 3 per group). E , F Thin layer chromatography images indicating shPISD treated cells had reduced mitochondrial PE content ( E ) and densitometry quantification of thin layer chromatography images presented in E ( n = 3 per group) ( F ). G A Seahorse mitochondrial stress test reveals shPISD treated cells have reduced FCCP stimulated respiration rates ( n = 10 per group). H shPISD treated cells have elevated proton leak, calculated by subtracting oxygen consumption rates (OCR) during basal conditions from OCR during oligomycin (oligo) treated conditions ( n = 10 per group). I , J Western blot image indicating shPISD treated cells have reduced complex I and IV protein abundance ( I ) and densitometry quantification of Western blot images shown in I ( n = 6 per group) ( J ). K 1- 14 C oleate oxidation is reduced in shPISD treated cells ( n = 6 per group). L 1- 14 C lignoceric acid oxidation is increased in shPISD treated cells ( n = 6 per group). M PCA plot of untargeted metabolomics data illustrating that shPISD treated cells have an altered metabolome compared to shSCR treated cells ( n = 3 per group). N Volcano plot of untargeted metabolomics data illustrating differences in metabolites between shSCR and shPISD treated cells ( n = 3 per group). O Heatmap of select lipid metabolism related metabolites that were differentially expressed between shSCR and shPISD treated cells ( n = 3 per group). The data are presented as means ± S.D.

    Journal: Oncogenesis

    Article Title: Targeting mitochondrial phosphatidylethanolamine alters mitochondrial metabolism and proliferation in hepatocellular carcinoma

    doi: 10.1038/s41389-025-00593-y

    Figure Lengend Snippet: A Schematic created using BioRender illustrating PISD enzyme reaction with phosphatidylserine (PS) to form CO 2 and phosphatidylethanolamine (PE). B Schematic created using BioRender showing generation of shScrambled (shSCR) and shPISD lentivirus in 293T cells and subsequent infection and gene silencing in HEPA1-6 cells. C The shPISD lentivirus reduced PISD mRNA abundance ( n = 2 per group) and altered the abundance of various genes involved in phospholipid metabolism ( n = 5 per group). D PISD activity was reduced in shPISD treated cells ( n = 3 per group). E , F Thin layer chromatography images indicating shPISD treated cells had reduced mitochondrial PE content ( E ) and densitometry quantification of thin layer chromatography images presented in E ( n = 3 per group) ( F ). G A Seahorse mitochondrial stress test reveals shPISD treated cells have reduced FCCP stimulated respiration rates ( n = 10 per group). H shPISD treated cells have elevated proton leak, calculated by subtracting oxygen consumption rates (OCR) during basal conditions from OCR during oligomycin (oligo) treated conditions ( n = 10 per group). I , J Western blot image indicating shPISD treated cells have reduced complex I and IV protein abundance ( I ) and densitometry quantification of Western blot images shown in I ( n = 6 per group) ( J ). K 1- 14 C oleate oxidation is reduced in shPISD treated cells ( n = 6 per group). L 1- 14 C lignoceric acid oxidation is increased in shPISD treated cells ( n = 6 per group). M PCA plot of untargeted metabolomics data illustrating that shPISD treated cells have an altered metabolome compared to shSCR treated cells ( n = 3 per group). N Volcano plot of untargeted metabolomics data illustrating differences in metabolites between shSCR and shPISD treated cells ( n = 3 per group). O Heatmap of select lipid metabolism related metabolites that were differentially expressed between shSCR and shPISD treated cells ( n = 3 per group). The data are presented as means ± S.D.

    Article Snippet: 1- 14 C Lignoceric Acid was purchased from American Radiolabeled Chemicals (ARC-0805-50, Saint Louis, MO, USA).

    Techniques: Infection, Activity Assay, Thin Layer Chromatography, Western Blot, Quantitative Proteomics